Manufacturer: Lasitan Substance: Furosemide (Lasix) Pack: 40mg (15 pills)
Lasix is a diuretic, which is an anthranilic acid derivative. Lasix tablets for oral administration contain furosemide as an active ingredient and inactive ingredients: NF lactose monohydrate, magnesium stearate NF, NF starch, USP talc and colloidal silicon dioxide NF. Chemically, this is 4-chloro-N-furfuryl-5-sulfamoylanthranilic acid.
Lasix is available as white tablets for prescription inside preparations in dosage strengths of 20, 40 and 80 mg. Furosemide is a white, creamy, crystalline powder, odorless. It is practically insoluble in water, sparingly soluble in alcohol, freely soluble in dilute alkaline solutions and insoluble in dilute acids.
Edematous syndrome of various genesis, incl. in chronic heart failure stage II-III, liver cirrhosis (portal hypertension syndrome), nephrotic syndrome. Pulmonary edema, cardiac asthma, brain edema, eclampsia, forced diuresis, hypertension, severe ways, some forms of hypertensive crisis, hypercalcemia.
Set individually, depending on the evidence, the clinical situation, the patient’s age. In the cycle of treatment, the dosage regimen is adjusted depending on the size of the diuretic response and the dynamics of the patient’s condition.
- When ingestion, the initial dose for adults is 20-80 mg / day, then, if necessary, the dose is gradually increased to 600 mg / day. For children, a single dose is 1-2 mg / kg.
- The maximum oral dose for children is 6 mg / kg.
- With the / in (jet) or in / m injection dose for adults is 20-40 mg 1 time / day, in some cases – 2 times / day. For children, the initial daily dose for parenteral use is 1 mg / kg.
In patients with cirrhosis and ascites, Lasix therapy in the hospital is best initiated. In the liver of Coma and the states, electrolyte depletion therapy should not be taken until the underlying condition has improved. Sudden changes in the balance of fluid and electrolytes in patients with cirrhosis can precipitate hepatic coma; Thus, strict observation during diuresis is necessary.
Additional potassium chloride and, if necessary, aldosterone antagonists in the prevention of hypokalemia and metabolic alkalosis. If the increase in azotemia and oliguria occurs during the treatment of severe progressive kidney disease, Lasix should be discontinued.